Linköping 2007 Anna-Lotta Hallbeck M. D. Studies of Transforming Growth Factor Alpha in Normal and Abnormal Growth

(A–D) Arrows mark Pdgfa lacZ -positive cells and arrowheads indicate extracellular X-gal staining; (E) arrows mark X-gal staining in bronchial epithelium and AEC2s, and arrowheads

Glycosaminoglycan GATA binding protein 3 Genetically engineered mouse model Gain-of-function Granulocyte-macrophage colony stimulating factor G-protein coupled receptor 77 Gene

lyzed from crude SF cell samples, was detected in all patients tested (6/6). The amount of TGF-alpha measured in SF did not correlate to the relative abun- dance of either cell

We discuss how the actions of TGF-bs and BMPs on cancer stem cells may explain their effects on tumor progression, and try to highlight intricate mechanisms that may link tumor

regulation by enhancing or suppressing expression or gene insertion (tumor suppressors, apoptosis- inducing genes, etc.) targeting cancer cells are in a growing number of

This study used the zebrafish xenograft assay by inject- ing malignant breast cancer cells into the embryonic circulation, and monitoring their invasion into the avascu- lar

During normal retinal vascularization, astrocyte precursor cells migrate from the optic nerve to populate the inner portion of the retina, where they form a scaffold on which

Ambulation in the morning was found to influence fasting GH values in different ways in young men and women; in the men, the values were lower compared to the resting state,