• No results found

Paracrine control of glucagon secretion in the pancreatic α-cell

N/A
N/A
Protected

Academic year: 2022

Share "Paracrine control of glucagon secretion in the pancreatic α-cell"

Copied!
1
0
0

Loading.... (view fulltext now)

Full text

(1)

DOCTORAL THESIS

SAHLGRENSKA ACADEMY

2020

Paracrine control of glucagon secretion in the pancreatic α-cell

Studies involving optogenetic cell activation

Caroline Miranda

ISBN 978-91-7833-952-5 (PRINT) ISBN 978-91-7833-953-2 (PDF) Printed by Stema Specialtryck AB, Borås

Paracrine control of glucagon secretion in the pancreatic α-cell | Caroline Miranda

SAHLGRENSKA ACADEMY

INSTITUTE OF NEUROSCIENCE AND PHYSIOLOGY

References

Related documents

The breast cancer microen vironment and cancer cell secretion | Emma P ersson.

Abbreviations: ACE model, model using additive genetic influence, shared environment (household effect), and the unique environment; AE model, model using additive genetic

Since the changes (and significant differ- ences) in blood flow and vascular conductance mirrored one another, we only discuss the blood flow values further. There were no

Tagged α‑Syn is Directed to EV‑Mediated Secretion To assess the distribution of α-syn after expression of the different tagged constructs, fractions separated with

Cell specificity of Ca 2+ response to tolbutamide in single ß-cells within intact islets We studied responses from individual ß-cells on the periphery of Fluo-3-labeled

Compared to b-cell factors, a conceptual advantage of somatostatin-mediated glu- cose inhibition of glucagon release is that it may operate also in hypoglycaemia, since

In islets exposed to 1 mM glucose, photoactivation of δ-cells transiently hyperpolarized α-cells, produced a long-lasting inhibition of glucagon exocytosis and

Furthermore, IL-6 and IL-8 are well-known to affect the cancer stem cell propagation [76, 147, 179] and induced secretion of these cytokines could partially be responsible for