Identification of Candidate Serum Proteins for Classifying Well-Differentiated Small Intestinal Neuroendocrine Tumors
Full text
Related documents
MDM2, PPM1D, located in the gain region on chromosome arm 17q, and CDKN2A have all been implicated in TP53 inactivation. TP53 is an
Administering the pan-PHD inhibitor DMOG in advanced stages of tumor development re- sulted in a significantly increased relative liver weight, a slight decrease in HCC and a minor
This is in line with previous and subsequent studies showing loss of chromosome 18 in 61-100% (Cunningham et al. In six cases loss of chromosome 18 was the only SCNA
Keywords: small intestine neuroendocrine tumor, somatic copy number alteration, expression profiling, APLP1, survival,
synaptophysin (small synaptic-like vesicles (27)) and chromogranin A (large dense-core vesicles (28)) (26). To ensure an epithelial phenotype cytokeratin is also often
and to differentiate true biological changes. An inappropriate normalization could induce misleading effects and thus affect the conclusions drawn from
SI-NET and LC patients’ clinical workup has been significantly improved during the last few decades. However, these malignancies have usually metastasized at diagnosis. This
ISBN 978-91-7833-364-6 (PRINT) ISBN 978-91-7833-365-3 (PDF) Printed by BrandFactory, Gothenburg. Small intestinal neuroendocrine tumours | T